As per recent reports, it has been revealed by a team of scientists, which had one expert of Indian origin, that reduced and poor lung function along with obstructive airway ailments can powerfully and alone result in terribly spiking up the risk of heart failure.

Importantly, as revealed by the researchers, the alliance was even obvious in people who never smoked and was still obvious following adjustments made in smoking status as well as in the number of years spent with smoking. This, they further claimed, clearly demonstrates "that our results are not primarily confounded by smoking".

The results were harvested from the study of Atherosclerosis Risk in Communities (ARIC), which was a cohort of population-origin from the United States of America, wherein a minimum of 16,000 adults between 45 and 64 years of age were tracked for a middling 15 years.

The study revealed that the long-term threat of falling prey to heart failure went up massively with a reduction in lung function as gauged by Forced Expiratory Volume (FEV1) by Spirometry, observations that were not changed on the basis of age, history of heart disease, or factors of cardiovascular damage, counting smoking.

The investigators also acknowledged the fact that Chronic Obstructive Pulmonary Disease (COPD) is a widespread and common co-morbidity among patients having heart failure, and also vice versa.

While expressing his views in this regard, along with mentioning what all consequences it can have among patients in the time to follow, Dr. Sunil Agarwal, the first author of the study from the Chapel Hill, USA-based University of North Carolina, said: "The public health implications are huge, particularly since smoking and air pollution affect lung function adversely. So it will be important to determine whether interventions that sustain or improve FEV1 are associated with lower risk of heart failure".